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Additional resources for Anxiety and Substance Use Disorders: The Vicious Cycle of Comorbidity (Series in Anxiety and Related Disorders)
R. (1986). A unified biosocial theory of personality and its role in the development of anxiety states. Psychiatric Developments, 3, 167–226. , et al. (1995). Is there a pathophysiological and therapeutic link between panic disorder and epilepsy? Acta Psychiatrica Scandanavica, 91, 430–432. Egan, M. , Kirch, D. , & Wyatt, R. J. (1994). Effects of chronic cocaine treatment on rat brain: Long- term reduction in frontal cortical serotonin. Biological Psychiatry, 36, 637–640. Eysenck, H. , & Eysenck, S.
The theory also allows for more general effects. For example, monoamines (serotonin, noradrenaline and dopamine) innervate much of the fear and anxiety systems. As a result, tricyclic drugs, clomipramine, monoamine oxidase inhibitors and selective serotonin reuptake inhibitors all increase monoamine function and so have both anti-anxiety and anti-panic effects. ) Likewise, many of the structures of the anxiety system (right hand side of Fig. 1) show coordinated rhythmic electrical activity (‘‘theta rhythm’’) controlled from subcortical areas such as the hypothalamus.
32 N. , & Coop, C. F. (1991). Neurochemically dissimilar anxiolytic drugs have common effects on hippocampal rhythmic slow activity. Neuropharmacology, 30, 855–863. , & Corr, P. J. (2004). A two-dimensional neuropsychology of defense: Fear/ anxiety and defensive distance. Neuroscience and Biobehavioral Reviews, 28, 285–305. , Panickar, K. , & Logan, B. (1996). The pituitary-adrenal axis and the different behavioral effects of buspirone and chlordiazepoxide. Pharmacology,Biochemistry and Behavior, 54, 51–56.